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| | Action Saunas: Saunas, Infra Red Saunas, Far Infrared Sauna, Home Saunas |
 | | Vasodilatation is also produced by axonal reflexes and by reflexes that change vasomotor balance. | | | Heating of one area of the body produces reflex-modulated vasodilatation in distant-body areas, even in the absence of a change in core body temperature; i.e. | | | Whole-body hyperthermia, with a consequent core temperature elevation, further induces vasodilatation via a hypothalamic-induced decrease in sympathetic tone on the arteriovenous anastomoses. |
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http://actionsaunas.com/painrelief.html
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| | Potassium channels mediate dilatation of cerebral arterioles in response to arachidonate -- Sobey et al. 275 (5): 1606 ... |
| | Mechanisms of impaired endothelium-dependent cerebral vasodilatation in response to bradykinin in hypertensive rats. | | | Vasodilatation in response to arachidonate was inhibited by catalase (100 U/ml, left ; n = 6 rats), superoxide dismutase (SOD, 100 U/ml, middle ; n = 6 rats), or deferoxamine (Deferox, 1 mM, right, n = 8 rats). | | | Vasodilatation is expressed as percent increase over baseline values, which were measured immediately before the agonists |
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http://ajpheart.physiology.org/cgi/content/full/275/5/H1606
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| | British Journal of Pharmacology - Different modulation by Ca2+-activated K+ channel blockers and herbimycin of ... |
| | The present study addressed whether endothelium-dependent vasodilatation evoked by acetylcholine and flow are mediated by the same mechanisms in isolated rat mesenteric small arteries, suspended in a pressure myograph for the measurement of internal diameter. | | | The aim of the present study was to clarify whether acetylcholine- and flow-induced vasodilatation is mediated by the same mechanisms in rat mesenteric small arteries. | | | Therefore, the thromboxane analog, U46619, was applied in the present study and this could be a limitation, since the processes leading to vasoconstriction evoked by the contractile drugs seem to play a role for relaxations mediated by endothelium-derived relaxing factors ( Plane and Garland, 1996 ; Tomioka et al., 1999). |
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http://www.nature.com/bjp/journal/v138/n8/full/0705214a.html
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| | ME Research Group for Education and Support - Research |
| | Conclusions: Prolongation of ACh-induced vasodilatation is suggestive of a disturbance to cholinergic pathways, perhaps within the vascular endothelium of patients with CFS, and might be related to some of the unusual vascular symptoms, such as hypotension and orthostatic intolerance, which are characteristic of the condition. | | | There are several possible ways in which increased vasodilatation to acetylcholine could be mediated, and this study represents further investigations into the phenomenon. | | | No differences in peak blood flow were seen between patients and controls, but the time taken for the ACh response to recover to baseline was significantly longer in the CFS patients than in control subjects. |
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http://www.meresearch.org.uk/research/sponsored/ach_prolong.html
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| | Life is but a headache! |
| | The vasodilatation response is readily learned by most patients, and the addition of the | | | By her third session she had developed control of her vasodilatation response, and by the fifth session she reported a significant decrease in Migraines. | | | She was instructed to begin exploring the sensations required to breathe from her diaphragm and to elevate her skin temperature. |
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http://www.brainwavecenter.com/library/headache/art_conn6.htm
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| | NO- and non-NO-, non-prostanoid-dependent vasodilatation in rat sciatic nerve during maturation and developing ... |
| | Thus, in biovascular preparations, SIN-1-induced vasodilatation is not mediated by peroxynitrite but by the NO-cGMP pathway. | | | Acetylcholine-induced vasodilatation in rat sciatic nerve microcirculation is dependent upon three vasodilators: NO, a vasodilatory prostanoid, and a NOS- and COX-independent K | | | In the present study, we examined the mechanisms of diabetic endothelial dysfunction in more detail by studying NO-related and non-NO-related pathways of agonist-induced vasodilatation. |
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http://jp.physoc.org/cgi/content/full/543/3/977
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| | Characterization of pulsed-dye laser-mediated vasodilatation |
| | Vasoconstriction is a clinical problem associated with invasive vascular procedures, microvascular reconstruction and subarachnoid hemorrhage. | | | Delivery of pulsed-dye laser energy has potential clinical application and warrants further investigation. | | | These studies provide characterization of pulsed-dye laser-mediated vasodilatation in an in vivo model. |
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http://omlc.ogi.edu/pubs/abs/schwengel93.html
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| | Viagra - In depth information about Viagra (Sildenafil Citrate), usage of Viagra and Success Stories |
| | This article is written to appropriately caution and not to unduly alarm physicians in their use of sildenafil in patients with heart disease. | | | The efficacy of sildenafil in enabling patients with erectile dysfunction due to a broad spectrum of causes, including vasculogenic (diabetes), neuroreflexogenic (spinal cord injury), and psychogenic (nonorganic), to achieve and maintain erection sufficient for satisfactory sexual intercourse has been demonstrated in all 21 double-blind, randomized, placebo-controlled, multicenter studies (Pfizer, unpublished data). | | | This is an area of particular concern for the patient with congestive heart failure who has a borderline low blood volume and a low blood pressure status as well as for the patient who is following a complicated, multidrug, antihypertensive therapy regimen. |
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http://www.abouthealthylife.com
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| | Prolonged aspirin inhibition of anodal vasodilation is not due to the trafficking delay of neural mediators -- Durand ... |
| | Spatial variability of vasodilatation in human forearm skin. | | | Durand S, Fromy B, Bouyé P, Saumet JL, and Abraham P. Vasodilatation in response to repeated anodal current application in the human skin relies on aspirin-sensitive mechanisms. | | | Durand S, Fromy B, Humeau A, Sigaudo-Roussel D, Saumet JL, and Abraham P. Break excitation alone does not explain the delay and amplitude of anodal current-induced vasodilatation in human skin. |
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http://ajpregu.physiology.org/cgi/content/full/285/1/R155
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| | Acetylcholine Mediated Vasodilatation in the Microcirculation of Patients with Chronic Fatigue Syndrome |
| | Phospholipase A2 activated prostacyclin (PGI2) also contributes to vasodilatation, and the contribution of this pathway to total relaxation can be determined by blocking with cyclo-oxygenase inhibitors such as aspirin. | | | Under normal circumstances with an intact endothelium it is thought that the calcium-activated NO pathway is responsible for the major component of acetylcholine-mediated vasodilatation. | | | Prolonged acetylcholine-induced vasodilatation in the peripheral microcirculation of patients with chronic fatigue syndrome. |
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http://www.chronicfatiguesupport.com/library/showarticle.cfm/id/5605
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| | [No title] |
| | Histamine seems to be important in the early phase (0-20 min) of these responses in the airways, while cyclo-oxygenase products (possibly PGD2) may be responsible for the long-lasting component. | | | Capsaicin pretreatment effectively desensitizes against local challenges with capsaicin in the airways and skin, as revealed by the absence of vasodilatory response 2 days later. | | | This may explain the symptoms experienced by PAF and MSA patients after alcohol. |
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http://www.webshells.com/medsrch/alcdilat.txt
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| | World Anaesthesia Issue 15 |
| | Prophylactic caffeine in such patients may reduce incidence of headache or simply try a cup of coffee in the postoperative period if not fasting. | | | Steroids, 5HT antagonists such as ondansetron, metronidazole, acetazolamide and muscle relaxants also may precipitate headache. | | | Dehydration causes traction on venous sinuses; hypoglycaemia leads to cerebral vasodilatation; caffeine normally induces vasoconstriction, acute withdrawal in those with high daily intake will cause rebound vasodilatation and headache. |
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http://www.nda.ox.ac.uk/wfsa/html/u15/u1514_01.htm
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| | Cardiovascular Science |
| | Background: It is well established that the free radical superoxide anion reacts with and destroys the vasodilator activity of endothelium-derived NO. More recently, work from our own laboratory has demonstrated that the Cu/Zn isoform of endogenous SOD is vitally important in protecting endothelium- and nitrergic nerve-derived NO from destruction by superoxide anion. | | | Recent work from our laboratory demonstrates that a major endogenous scavenger of superoxide, the Cu/Zn isoform of superoxide dismutase (SOD), exerts a tonic protective action on nitric oxide following its release from the vascular endothelium 1, 2 or nitrergic nerves 3,45. | | | Moreover, a number of cardiovascular pathologies including hypertension, atherosclerosis, ischaemia-reperfusion injury, heart failure and stroke are associated with impaired NO-mediated vasodilatation, resulting from enhanced activity of superoxide anion. |
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http://www.gla.ac.uk/departments/ibls/GradSchool/2005/cardio.htm
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| | Northeast Rehabilitation Health Network |
| | Red blotches on the skin above level of spinal injury (secondary to vasodilatation) | | | Sweating above level of spinal injury (secondary to vasodilatation) | | | Bradycardia - slow pulse <60 beats per minute - (secondary to vagal parasympathetic stimulation) |
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http://www.northeastrehab.com/Articles/dysreflexia.htm
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| | [No title] |
| | The present study suggests that Ca2+-activated K+ channels are involved in acetylcholine—evoked vasodilatation, while a Src tyrosine kinase is involved in flow-induced nitric oxide (NO)-mediated vasodilatation in rat mesenteric small arteries. | | | Inhibition of Ca2+-activated K+ channels with the combination of apamin and charybdotoxin did not change shear stress-and acetylcholine-evoked vasodilatation. | | | Vessels were contracted with the tromboxane analogue, U46619 (0.1 (M) and flow was applied by a peristaltic pump resulting in shear stress levels of 4 and 16 dyn/cm2. |
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http://www.hypertension.suite.dk/kalender/arkiv/2003/nr14.doc
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| | News - Angiotensin-Converting Enzyme Inhibitors Increase Peripheral Vasodilatation And T-Pa Release |
| | Fraser N. Witherow and colleagues from the University of Edinburgh enrolled nine patients with symptomatic heart failure arising from ischaemic heart disease who were undergoing chronic treatment with ACE inhibitors. | | | The authors concluded that long-term ACE inhibitor treatment increases peripheral vasodilatation and local t-PA release induced by bradykinin in patients with heart failure arising from ischaemic heart disease. | | | Previous studies suggested that bradykinin, a potent endothelial cell stimulant, triggers vasodilatation and t-PA release. |
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http://www.docguide.com/news/content.nsf/news/8525697700573E1885256C3700557484
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| | APStracts 8:0371H, 2001. |
| | In contrast, systemic theophylline, which readily penetrates the BBB, nearly abolished the SNS-induced vasodilatation (P < 0.01; n = 7). | | | Hypercarbic vasodilatation and somatosensory evoked potentials were not affected by any of the compounds tested. | | | The pial circulation was observed through a closed cranial window in a-chloralose-anesthetized rats. |
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http://www.uth.tmc.edu/apstracts/2001/heart/July/371h.html
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| | MRI NO2 Review by sucram |
| | BACKGROUND: The role of endothelin (ET)-1 in maintenance of basal vascular tone has been demonstrated by local and systemic vasodilatation to endothelin receptor antagonists in humans. | | | The study focuses on nitric oxcide synthesis and its effect on vasodilatation. | | | Although the constrictor effects mediated by the vascular smooth muscle ET(A) receptors are clear, the contribution from endothelial and vascular smooth muscle ET(B) receptors remains to be defined. |
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http://www.goendurance.com/z3173_review_NO2_MRI.html
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