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| | Force-velocity and power-load curves in rat skinned cardiac myocytes -- McDonald et al. 511 (2): 519 -- The Journal of ... |
 | | This study utilized a skinned myocyte preparation with low end compliance to examine force-velocity and power-load curves at 12 °C in myocytes from rat hearts. |  | | Force-velocity relationships were determined in skinned myocyte preparations by measuring shortening velocities during force clamps imposed by a servo-controlled feedback system (Sweitzer and Moss, 1993). |  | | Myocytes were obtained by mechanical disruption of hearts from Sprague-Dawley rats. |
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http://jp.physoc.org/cgi/content/full/511/2/519
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| | Ablation of telomerase and telomere loss leads to cardiac dilatation and heart failure associated with p53 upregulation |
 | | These parameters were determined with the expectation that telomeric shortening and attenuation in cardiac myocyte division as a function of generation and age characteristically led to enhanced myocyte apoptosis, reactive hypertrophy, restructuring of the wall and depression of ventricular hemodynamics. |  | | It is a rather common event that the development of heart failure mediated by chronic cardiac myocyte loss is rapid and may evolve quickly into death of the organism, suggesting a cardiac dysfunction phenotype in G5 Terc |  | | Recently, this notion has been challenged and evidence for cardiac myocyte replication has been obtained in animals and humans ( Kajstura et al., 1998 ; Beltrami et al., 2001 ; Anversa and Nadal-Ginard, 2002 ; Quaini et al., 2002). |
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http://www.nature.com/emboj/journal/v22/n1/full/7594923a.html
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| | Indian Pacing and Electrophysiology Journal - Zaugg |
 | | Thereafter, the vicious circle of myocyte Ca overload is established and therapeutic attempts have an inherently lower likelihood of success. |  | | Further contributions to postfibrillatory myocardial dysfunction may arise from ischemia-induced oxygen free radicals and/or from the impact of electrical defibrillation shocks. |  | | Therefore, postfibrillatory myocardial dysfunction presumably is a consequence of reduced myofilament Ca responsiveness following myocyte Ca overload caused by both VF and consequent ischemia. |
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http://www.ipej.org/0402/zaugg.htm
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| | ICU CARDIOVASCULAR PAGE, hosted & composed by LARA & Johan |
 | | The ferocious utilization of energy by the myocyte is also reflected in the abundant capillaries coursing through the myocardium, and the large amount of myoglobin within heart muscle. |  | | A lot of this is however people boasting about what they have done (without specifics) or saying what they are about to do. |  | | Cardiac myocytes are packed with mitochondria, to provide the energy for the perhaps two billion heartbeats that will occur during a human lifespan. |
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http://www.anaesthetist.com/icu/organs/heart/myocyte.htm
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| | IonOptix Myocyte Systems |
 | | Myocyte Systems are offered with any of our three excitation light sources: the HyperSwitch, the StepperSwitch, or the Optoscan. |  | | Due to the great popularity of these systems, we are also supplying a number of peripheral items one may need when setting up a new system such as chambers, heaters, perfusion systems. |  | | Below are links to pages for the individual components that make up our Myocyte Systems. |
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http://www.ionoptix.com/web1/body/myo_sy.htm
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| | myocyte TutorGig.co.uk Encyclopedia |
 | | There are three types of muscle cells found in the three types of muscle tissue; skeletal muscle, cardiac muscle, and smooth muscle. |  | | Cardiac Myocyte: Connective Tissue Interactions in Health and Disease.. |  | | 'Myocyte ' is the technical term for a muscle cell. |
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http://www.tutorgig.co.uk/ed/myocyte
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| | Myocyte Death in Streptozotocin-Induced Diabetes in Rats Is Angiotensin II- Dependent |
 | | Thus diabetic cardiomyopathy may be viewed as an AT II-dependent process in which that peptide plays a critical role in myocyte death and hypertrophy. |  | | Myocyte Death in Streptozotocin-Induced Diabetes in Rats Is Angiotensin II- Dependent |  | | Angiotensinogen, renin, and AT receptor increased in myocytes from diabetic rat hearts, while angiotensin-converting enzyme and AT remained constant. |
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http://info.med.yale.edu/labinvest/abstracts/00months/0004Apr/0004_513.html
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| | Myocyte Death and Growth in the Failing Heart |
 | | I. Pathology of Heart Failure A. Recognition of myocyte death B.Myocyte death and heart failure II. |  | | p53 and Bcl-2 Family of Proteins in Heart Failure III.Myocyte Hypertrophy and Heart Failure A. Side-to-side slippage of myocytes and heart failure B. Myocyte proliferation and heart failure IV. |  | | Myocyte Death and Growth in the Failing Heart |
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http://info.med.yale.edu/labinvest/abstracts/98months/9807jul/0798_002.html
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